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Lodato, MichaelPerson Why?
SINGLE-CELL ANALYSIS OF SOMATIC MUTATION IN AGING ANO NEUROOEGENERATIVE DISEASE IN THE HUMAN BRAINGrant Why?
Somatic mutation in single human neurons tracks developmental and transcriptional history.Academic Article Why?
DNA polymerases ß and ? do not directly affect Ig variable region somatic hypermutation although their absence reduces the frequency of mutations.Academic Article Why?
Somatic mutations of epidermal growth factor receptor in colorectal carcinoma.Academic Article Why?
Hierarchy in somatic mutations arising during genomic evolution and progression of follicular lymphoma.Academic Article Why?
RUNX1-targeted therapy for AML expressing somatic or germline mutation in RUNX1.Academic Article Why?
More Frequent than Desired: Midgut Stem Cell Somatic Mutations.Academic Article Why?
Somatic mutations in histiocytic sarcoma identified by next generation sequencing.Academic Article Why?
Impact of somatic mutation on the S107(T15) heavy-chain V region of antibodies reactive with self and nonself.Academic Article Why?
Genome aging: somatic mutation in the brain links age-related decline with disease and nominates pathogenic mechanisms.Academic Article Why?
Concordance of somatic mutation profiles [BRAF, NRAS and TERT] and tumoral PD-L1 in matched primary cutaneous and metastatic melanoma samples.Academic Article Why?
Corrigendum: Genome aging: somatic mutation in the brain links age-related decline with disease and nominates pathogenic mechanisms.Academic Article Why?
Differential expression of APE1 and APE2 in germinal centers promotes error-prone repair and A:T mutations during somatic hypermutation.Academic Article Why?
Immunohistochemical loss of 5-hydroxymethylcytosine expression in acute myeloid leukaemia: relationship to somatic gene mutations affecting epigenetic pathways.Academic Article Why?
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