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One or more keywords matched the following properties of
Malaria, Plasmodium vivax
One or more keywords matched the following items that are connected to
Neutrophil paralysis in Plasmodium vivax malaria.
Malaria-induced NLRP12/NLRP3-dependent caspase-1 activation mediates inflammation and hypersensitivity to bacterial superinfection.
The CD14+CD16+ inflammatory monocyte subset displays increased mitochondrial activity and effector function during acute Plasmodium vivax malaria.
Induction of Inhibitory Receptors on T Cells During Plasmodium vivax Malaria Impairs Cytokine Production.
DNA-Containing Immunocomplexes Promote Inflammasome Assembly and Release of Pyrogenic Cytokines by CD14+ CD16+ CD64high CD32low Inflammatory Monocytes from Malaria Patients.
T follicular helper cells regulate the activation of B lymphocytes and antibody production during Plasmodium vivax infection.
Plasmodium vivax Infection Impairs Regulatory T-Cell Suppressive Function During Acute Malaria.
The immunology of Plasmodium vivax malaria.
Plasmodium vivax Infection Alters Mitochondrial Metabolism in Human Monocytes.
Asymptomatic Plasmodium vivax malaria in the Brazilian Amazon: Submicroscopic parasitemic blood infects Nyssorhynchus darlingi.
Malaria Plasmodium vivax