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Dendritic cells lose ability to present protein antigen after stimulating antigen-specific T cell responses, despite upregulation of MHC class II expression.
IL-6-gp130-STAT3 in T cells directs the development of IL-17+ Th with a minimum effect on that of Treg in the steady state.
Fast on-rates allow short dwell time ligands to activate T cells.
Viral antigen density and confinement time regulate the reactivity pattern of CD4 T-cell responses to vaccinia virus infection.
Thymic selection stifles TCR reactivity with the main chain structure of MHC and forces interactions with the peptide side chains.
Single naive CD4+ T cells from a diverse repertoire produce different effector cell types during infection.
Antigen affinity and antigen dose exert distinct influences on CD4 T-cell differentiation.
I-Ag7 ?56/57 polymorphisms regulate non-cognate negative selection to CD4+ T cell orchestrators of type 1 diabetes.
CD4 Effector TCR Avidity for Peptide on APC Determines the Level of Memory Generated.
CD4 Positive T Lymphocytes