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Highly cross-reactive T cell responses to myelin basic protein epitopes reveal a nonpredictable form of TCR degeneracy.
Single naive CD4+ T cells from a diverse repertoire produce different effector cell types during infection.
Hydrophobic CDR3 residues promote the development of self-reactive T cells.
Affinity for self antigen selects Treg cells with distinct functional properties.
Patients with CD3G mutations reveal a role for human CD3? in Treg diversity and suppressive function.
Resolving the Instructions for ?? T Cell Development.