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Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.
RIPK1 blocks early postnatal lethality mediated by caspase-8 and RIPK3.
RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosis.
CYLD Proteolysis Protects Macrophages from TNF-Mediated Auto-necroptosis Induced by LPS and Licensed by Type I IFN.
RIPK1 and RIPK3 Kinases Promote Cell-Death-Independent Inflammation by Toll-like Receptor 4.
The pseudokinase MLKL activates PAD4-dependent NET formation in necroptotic neutrophils.
Pathogen blockade of TAK1 triggers caspase-8-dependent cleavage of gasdermin D and cell death.
Elevated A20 promotes TNF-induced and RIPK1-dependent intestinal epithelial cell death.
RIPK1 Mediates TNF-Induced Intestinal Crypt Apoptosis During Chronic NF-?B Activation.
Ptpn6 inhibits caspase-8- and Ripk3/Mlkl-dependent inflammation.