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Landers, John
One or more keywords matched the following items that are connected to
Landers, John
Item Type
Name
Academic Article
A common haplotype within the PON1 promoter region is associated with sporadic ALS.
Academic Article
Chromosome 9p21 in sporadic amyotrophic lateral sclerosis in the UK and seven other countries: a genome-wide association study.
Academic Article
Mapping of gene expression reveals CYP27A1 as a susceptibility gene for sporadic ALS.
Academic Article
Age of onset of amyotrophic lateral sclerosis is modulated by a locus on 1p34.1.
Academic Article
Reduced expression of the Kinesin-Associated Protein 3 (KIFAP3) gene increases survival in sporadic amyotrophic lateral sclerosis.
Academic Article
Genome-wide association study identifies 19p13.3 (UNC13A) and 9p21.2 as susceptibility loci for sporadic amyotrophic lateral sclerosis.
Academic Article
A large genome scan for rare CNVs in amyotrophic lateral sclerosis.
Concept
Polymorphism, Single Nucleotide
Academic Article
The C9ORF72 expansion mutation is a common cause of ALS+/-FTD in Europe and has a single founder.
Academic Article
No evidence for shared genetic basis of common variants in multiple sclerosis and amyotrophic lateral sclerosis.
Academic Article
Association of a Locus in the CAMTA1 Gene With Survival in Patients With Sporadic Amyotrophic Lateral Sclerosis.
Academic Article
Genome-wide association study identifies susceptibility loci for open angle glaucoma at TMCO1 and CDKN2B-AS1.
Academic Article
Common variants near ABCA1, AFAP1 and GMDS confer risk of primary open-angle glaucoma.
Academic Article
Genome-wide association study of intraocular pressure uncovers new pathways to glaucoma.
Academic Article
Meta-analysis of genome-wide association studies identifies novel loci that influence cupping and the glaucomatous process.
Academic Article
Multitrait analysis of glaucoma identifies new risk loci and enables polygenic prediction of disease susceptibility and progression.
Academic Article
Genetic analysis of amyotrophic lateral sclerosis identifies contributing pathways and cell types.
Academic Article
Loss of function mutations in GEMIN5 cause a neurodevelopmental disorder.
Search Criteria
Polymorphism Single Nucleotide