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FUS/TLS assembles into stress granules and is a prosurvival factor during hyperosmolar stress.
Amyotrophic lateral sclerosis-linked FUS/TLS alters stress granule assembly and dynamics.
Autophagy meets fused in sarcoma-positive stress granules.
Quantitative proteomics identifies proteins that resist translational repression and become dysregulated in ALS-FUS.