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dSarm/Sarm1 is required for activation of an injury-induced axon death pathway.
Reduced expression of the Kinesin-Associated Protein 3 (KIFAP3) gene increases survival in sporadic amyotrophic lateral sclerosis.
The distinct genetic pattern of ALS in Turkey and novel mutations.
Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1.
Genome-wide association analyses identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis.
Further analysis of KIFAP3 gene in ALS patients from Switzerland and Sweden.
Loss of Sarm1 does not suppress motor neuron degeneration in the SOD1G93A mouse model of amyotrophic lateral sclerosis.
Genetic diversity of axon degenerative mechanisms in models of Parkinson's disease.