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Endotoxin tolerance dysregulates MyD88- and Toll/IL-1R domain-containing adapter inducing IFN-beta-dependent pathways and increases expression of negative regulators of TLR signaling.
Inflammation and fibrosis during Chlamydia pneumoniae infection is regulated by IL-1 and the NLRP3/ASC inflammasome.
The NLRP3 inflammasome is up-regulated in cardiac fibroblasts and mediates myocardial ischaemia-reperfusion injury.
The Toll-IL-1 receptor adaptor family grows to five members.
AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC.
The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses.
NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals.
Inflammation in mice ectopically expressing human Pyogenic Arthritis, Pyoderma Gangrenosum, and Acne (PAPA) Syndrome-associated PSTPIP1 A230T mutant proteins.
Synthetic oligodeoxynucleotides containing suppressive TTAGGG motifs inhibit AIM2 inflammasome activation.
Salmonella infection induces recruitment of Caspase-8 to the inflammasome to modulate IL-1? production.
Unified polymerization mechanism for the assembly of ASC-dependent inflammasomes.
Cell Survival and Cytokine Release after Inflammasome Activation Is Regulated by the Toll-IL-1R Protein SARM.