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NOD congenic mice genetically protected from autoimmune diabetes remain resistant to transplantation tolerance induction.
Genetic separation of the transplantation tolerance and autoimmune phenotypes in NOD mice.
Islet cell autoimmunity and transplantation tolerance: two distinct mechanisms?
Partial versus full allogeneic hemopoietic chimerization is a preferential means to inhibit type 1 diabetes as the latter induces generalized immunosuppression.
Genetic disassociation of autoimmunity and resistance to costimulation blockade-induced transplantation tolerance in nonobese diabetic mice.
Autoimmune diabetes and resistance to xenograft transplantation tolerance in NOD mice.