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Glucose transporter recycling in response to insulin is facilitated by myosin Myo1c.
Role of EHD1 and EHBP1 in perinuclear sorting and insulin-regulated GLUT4 recycling in 3T3-L1 adipocytes.
RNAi-based analysis of CAP, Cbl, and CrkII function in the regulation of GLUT4 by insulin.
Tumor necrosis factor alpha (TNFalpha) stimulates Map4k4 expression through TNFalpha receptor 1 signaling to c-Jun and activating transcription factor 2.
Akt substrate TBC1D1 regulates GLUT1 expression through the mTOR pathway in 3T3-L1 adipocytes.
Paradoxical effect of mitochondrial respiratory chain impairment on insulin signaling and glucose transport in adipose cells.
A phosphatidylinositol 3-kinase-independent insulin signaling pathway to N-WASP/Arp2/3/F-actin required for GLUT4 glucose transporter recycling.
C2 domain-containing phosphoprotein CDP138 regulates GLUT4 insertion into the plasma membrane.
Conventional kinesin KIF5B mediates insulin-stimulated GLUT4 movements on microtubules.
Unconventional myosin Myo1c promotes membrane fusion in a regulated exocytic pathway.
The v-SNARE Vti1a regulates insulin-stimulated glucose transport and Acrp30 secretion in 3T3-L1 adipocytes.
An RNA interference-based screen identifies MAP4K4/NIK as a negative regulator of PPARgamma, adipogenesis, and insulin-responsive hexose transport.
RNAi-based gene silencing in primary mouse and human adipose tissues.
Insulin stimulates membrane fusion and GLUT4 accumulation in clathrin coats on adipocyte plasma membranes.
The interaction of Akt with APPL1 is required for insulin-stimulated Glut4 translocation.
A novel pleckstrin homology domain-containing protein enhances insulin-stimulated Akt phosphorylation and GLUT4 translocation in adipocytes.
Glucose Transporter Type 1
Glucose Transporter Type 4
Glucose Transporter Type 2