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Anti-endothelial cell autoantibodies in BB rats with spontaneous and induced IDDM.
The T cell marker RT6 in a rat model of autoimmune diabetes.
Induction of immunological tolerance to islet allografts.
Treatment of allograft recipients with donor-specific transfusion and anti-CD154 antibody leads to deletion of alloreactive CD8+ T cells and prolonged graft survival in a CTLA4-dependent manner.
Autoimmune destruction of islets transplanted into RT6-depleted diabetes-resistant BB/Wor rats.
Costimulatory blockade induces hyporesponsiveness in T cells that recognize alloantigen via indirect antigen presentation.
Absence of RT6+ T cells in diabetes-prone biobreeding/Worcester rats is due to genetic and cell developmental defects.
Loss of RT6 message and most circulating T cells after thymectomy of diabetes prone BB rats.
Development of new-generation HU-PBMC-NOD/SCID mice to study human islet alloreactivity.
Biochemical studies of RT6 alloantigens in BB/Wor and normal rats. Evidence for intact unexpressed RT6a structural gene in diabetes-prone BB rats.
The RT6 rat lymphocyte alloantigen circulates in soluble form.