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The Bax subfamily of Bcl2-related proteins is essential for apoptotic signal transduction by c-Jun NH(2)-terminal kinase.
Interaction of the c-Jun/JNK pathway and cyclin-dependent kinases in death of embryonic cortical neurons evoked by DNA damage.
Survival signaling mediated by c-Jun NH(2)-terminal kinase in transformed B lymphoblasts.
Multisite phosphorylation regulates Bim stability and apoptotic activity.
Mcl-1 integrates the opposing actions of signaling pathways that mediate survival and apoptosis.
Functional cooperation of the proapoptotic Bcl2 family proteins Bmf and Bim in vivo.
JNK phosphorylation of Bim-related members of the Bcl2 family induces Bax-dependent apoptosis.
Proto-Oncogene Proteins c-bcl-2