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Modeling genome-wide replication kinetics reveals a mechanism for regulation of replication timing.
Mre11 nuclease activity and Ctp1 regulate Chk1 activation by Rad3ATR and Tel1ATM checkpoint kinases at double-strand breaks.
Signaling pathways that regulate cell division.
An intrinsic checkpoint model for regulation of replication origins.
The role of specific checkpoint-induced S-phase transcripts in resistance to replicative stress.
Replication timing and its emergence from stochastic processes.
The capacity of origins to load MCM establishes replication timing patterns.