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Differential effects of a Toll-like receptor antagonist on Mycobacterium tuberculosis-induced macrophage responses.
Characterization of proinflammatory cytokine production and CD14 expression by murine alveolar macrophage cell lines.
Pulmonary lipopolysaccharide (LPS)-binding protein inhibits the LPS-induced lung inflammation in vivo.
MUC1 mucin is a negative regulator of toll-like receptor signaling.