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Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9.
Malaria primes the innate immune response due to interferon-gamma induced enhancement of toll-like receptor expression and function.
Plasmodium falciparum infection causes proinflammatory priming of human TLR responses.
Innate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genome.
Innate sensing of malaria parasites.
Cyclic GMP-AMP Synthase Is the Cytosolic Sensor of Plasmodium falciparum Genomic DNA and Activates Type I IFN in Malaria.
Cutting Edge: Plasmodium falciparum Induces Trained Innate Immunity.
Targeting the IL33-NLRP3 axis improves therapy for experimental cerebral malaria.
Lymphocyte crosstalk is required for monocyte-intrinsic trained immunity to Plasmodium falciparum.