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Search Results to Douglas T Golenbock MD

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One or more keywords matched the following items that are connected to Golenbock, Douglas

Item TypeName
Academic Article Involvement of CD14 and complement receptors CR3 and CR4 in nuclear factor-kappaB activation and TNF production induced by lipopolysaccharide and group B streptococcal cell walls.
Academic Article Human toll-like receptor 2 mediates monocyte activation by Listeria monocytogenes, but not by group B streptococci or lipopolysaccharide.
Academic Article c-Jun kinase is a critical signaling molecule in a neonatal model of group B streptococcal sepsis.
Academic Article A mechanism for neurodegeneration induced by group B streptococci through activation of the TLR2/MyD88 pathway in microglia.
Academic Article Toll-like receptor-dependent discrimination of streptococci.
Academic Article TLR-independent type I interferon induction in response to an extracellular bacterial pathogen via intracellular recognition of its DNA.
Academic Article Role of p38 and early growth response factor 1 in the macrophage response to group B streptococcus.
Academic Article Novel engagement of CD14 and multiple toll-like receptors by group B streptococci.
Academic Article Activation of the NLRP3 inflammasome by group B streptococci.
Academic Article Cellular activation, phagocytosis, and bactericidal activity against group B streptococcus involve parallel myeloid differentiation factor 88-dependent and independent signaling pathways.
Academic Article Dual role of TLR2 and myeloid differentiation factor 88 in a mouse model of invasive group B streptococcal disease.
Academic Article Role of lipoteichoic acid in the phagocyte response to group B streptococcus.
Academic Article Lipoproteins are critical TLR2 activating toxins in group B streptococcal sepsis.
Academic Article Macrophages recognize streptococci through bacterial single-stranded RNA.
Academic Article NO is a macrophage autonomous modifier of the cytokine response to streptococcal single-stranded RNA.
Academic Article The Abi-domain protein Abx1 interacts with the CovS histidine kinase to control virulence gene expression in group B Streptococcus.
Concept Streptococcus agalactiae
Academic Article RNA and ß-hemolysin of group B Streptococcus induce interleukin-1ß (IL-1ß) by activating NLRP3 inflammasomes in mouse macrophages.

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  • Streptococcus agalactiae