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Networks and hubs for the transcriptional control of osteoblastogenesis.
Structural coupling of Smad and Runx2 for execution of the BMP2 osteogenic signal.
Specific residues of RUNX2 are obligatory for formation of BMP2-induced RUNX2-SMAD complex to promote osteoblast differentiation.
Runx2-Smad signaling impacts the progression of tumor-induced bone disease.
Expression of the IL-11 Gene in Metastatic Cells Is Supported by Runx2-Smad and Runx2-cJun Complexes Induced by TGF?1.