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Differential suppression of priA2::kan phenotypes in Escherichia coli K-12 by mutations in priA, lexA, and dnaC.
Overlapping functions for recF and priA in cell viability and UV-inducible SOS expression are distinguished by dnaC809 in Escherichia coli K-12.
Requirements for ATP binding and hydrolysis in RecA function in Escherichia coli.
UvrD303, a hyperhelicase mutant that antagonizes RecA-dependent SOS expression by a mechanism that depends on its C terminus.
RecOR suppression of recF mutant phenotypes in Escherichia coli K-12.
Use of high and low level overexpression plasmids to test mutant alleles of the recF gene of Escherichia coli K-12 for partial activity.
Measurement of SOS expression in individual Escherichia coli K-12 cells using fluorescence microscopy.
Localization of RecA in Escherichia coli K-12 using RecA-GFP.
RecA-mediated SOS induction requires an extended filament conformation but no ATP hydrolysis.
Single molecule analysis of a red fluorescent RecA protein reveals a defect in nucleoprotein filament nucleation that relates to its reduced biological functions.
Studies on the mechanism of reduction of UV-inducible sulAp expression by recF overexpression in Escherichia coli K-12.
recO and recR mutations delay induction of the SOS response in Escherichia coli.
An Epistasis Analysis of recA and recN in Escherichia coli K-12.