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Lack of a relation between human neonatal thyroxine and pediatric neurobehavioral disorders: neonatal total thyroxine is not a good proxy measure of maternal thyroid hormone insufficiency.
Polychlorinated biphenyls exert selective effects on cellular composition of white matter in a manner inconsistent with thyroid hormone insufficiency.
Maternal hypothyroidism selectively affects the expression of neuroendocrine-specific protein A messenger ribonucleic acid in the proliferative zone of the fetal rat brain cortex.
Polychlorinated biphenyls (Aroclor 1254) do not uniformly produce agonist actions on thyroid hormone responses in the developing rat brain.
Identification of thyroid hormone receptor binding sites and target genes using ChIP-on-chip in developing mouse cerebellum.
Polychlorinated biphenyls (PCBs) exert thyroid hormone-like effects in the fetal rat brain but do not bind to thyroid hormone receptors.
Mode of action: developmental thyroid hormone insufficiency--neurological abnormalities resulting from exposure to propylthiouracil.
The nature of the compensatory response to low thyroid hormone in the developing brain.
Thyroid hormone may regulate mRNA abundance in liver by acting on microRNAs.
Thyroid hormones regulate levels of thyrotropin-releasing-hormone mRNA in the paraventricular nucleus.
Maternal Thyroid Function During Pregnancy or Neonatal Thyroid Function and Attention Deficit Hyperactivity Disorder: A Systematic Review.
Fluoride exposure and hypothyroidism in a Canadian pregnancy cohort.