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Innate and acquired control of trypanosome parasitaemia in Cape buffalo.
Anti-Trypanosoma brucei activity in Cape buffalo serum during the cryptic phase of parasitemia is mediated by antibodies.
Induction and regulation of Trypanosoma brucei VSG-specific antibody responses.
T. brucei infection reduces B lymphopoiesis in bone marrow and truncates compensatory splenic lymphopoiesis through transitional B-cell apoptosis.
Trypanosomiasis-induced B cell apoptosis results in loss of protective anti-parasite antibody responses and abolishment of vaccine-induced memory responses.
Anti-Trypanosoma brucei activity of nonprimate zoo sera.
Infection-associated decline of cape buffalo blood catalase augments serum trypanocidal activity.