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Supraspinal input is dispensable to generate glycine-mediated locomotive behaviors in the zebrafish embryo.
Gfap-positive radial glial cells are an essential progenitor population for later-born neurons and glia in the zebrafish spinal cord.
Expressing acetylcholine receptors after innervation suppresses spontaneous vesicle release and causes muscle fatigue.
Zebrafish prdm12b acts independently of nkx6.1 repression to promote eng1b expression in the neural tube p1 domain.
GABAA a subunit control of hyperactive behavior in developing zebrafish.