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E2F mediates dihydrofolate reductase promoter activation and multiprotein complex formation in human cytomegalovirus infection.
The human cytomegalovirus IE1-72 protein interacts with the cellular p107 protein and relieves p107-mediated transcriptional repression of an E2F-responsive promoter.
The viral oncoprotein E1A blocks transforming growth factor beta-mediated induction of p21/WAF1/Cip1 and p15/INK4B.
E2F1-specific induction of apoptosis and p53 accumulation, which is blocked by Mdm2.
Active RB elicits late G1/S inhibition.
The APC tumor suppressor controls entry into S-phase through its ability to regulate the cyclin D/RB pathway.
Apoptosis associated with deregulated E2F activity is dependent on E2F1 and Atm/Nbs1/Chk2.
Life, death and E2F: linking proliferation control and DNA damage signaling via E2F1.
Compensation of BRG-1 function by Brm: insight into the role of the core SWI-SNF subunits in retinoblastoma tumor suppressor signaling.
Cellular targets for activation by the E2F1 transcription factor include DNA synthesis- and G1/S-regulatory genes.
Rb inactivation leads to E2F1-mediated DNA double-strand break accumulation.
E2F1 overexpression in quiescent fibroblasts leads to induction of cellular DNA synthesis and apoptosis.
Retinoblastoma-Like Protein p107
Retinoblastoma-Binding Protein 1