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One or more keywords matched the following items that are connected to Xu, Zuoshang
Item TypeName
Academic Article Mitochondrial electron transport chain complex dysfunction in a transgenic mouse model for amyotrophic lateral sclerosis.
Academic Article ALS-associated mutant SOD1G93A causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes.
Academic Article Pol II-expressed shRNA knocks down Sod2 gene expression and causes phenotypes of the gene knockout in mice.
Academic Article Overexpression of neurofilament subunit M accelerates axonal transport of neurofilaments.
Academic Article Transgenic RNAi: Accelerating and expanding reverse genetics in mammals.
Academic Article Allele-specific RNAi selectively silences mutant SOD1 and achieves significant therapeutic benefit in vivo.
Academic Article Temporal and spatial variations in slow axonal transport velocity along peripheral motoneuron axons.
Academic Article A quantitative histochemical assay for activities of mitochondrial electron transport chain complexes in mouse spinal cord sections.
Academic Article Mutant Cu, Zn superoxide dismutase that causes motoneuron degeneration is present in mitochondria in the CNS.
Academic Article A non-specific effect associated with conditional transgene expression based on Cre-loxP strategy in mice.
Academic Article Mitochondrial degeneration in amyotrophic lateral sclerosis.
Academic Article Inhibition of chaperone activity is a shared property of several Cu,Zn-superoxide dismutase mutants that cause amyotrophic lateral sclerosis.
Academic Article S-nitrosothiol depletion in amyotrophic lateral sclerosis.
Academic Article Widespread aggregation of mutant VAPB associated with ALS does not cause motor neuron degeneration or modulate mutant SOD1 aggregation and toxicity in mice.
Academic Article Identification of human monoclonal antibodies specific for human SOD1 recognizing distinct epitopes and forms of SOD1.
Concept Mice, Transgenic
Academic Article Mutant PFN1 causes ALS phenotypes and progressive motor neuron degeneration in mice by a gain of toxicity.
Academic Article Imaging Net Retrograde Axonal Transport In Vivo: A Physiological Biomarker.
Academic Article Protein citrullination marks myelin protein aggregation and disease progression in mouse ALS models.
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  • Mice Transgenic