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Single particle analysis of relaxed and activated muscle thin filaments.
The C terminus of cardiac troponin I stabilizes the Ca2+-activated state of tropomyosin on actin filaments.
Orientation of myosin binding protein C in the cardiac muscle sarcomere determined by domain-specific immuno-EM.
Skeletal myosin binding protein-C isoforms regulate thin filament activity in a Ca2+-dependent manner.
Altered C10 domain in cardiac myosin binding protein-C results in hypertrophic cardiomyopathy.
Amino terminus of cardiac myosin binding protein-C regulates cardiac contractility.