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Vaccinia peptides eluted from HLA-DR1 isolated from virus-infected cells are recognized by CD4+ T cells from a vaccinated donor.
HLA-DM constrains epitope selection in the human CD4 T cell response to vaccinia virus by favoring the presentation of peptides with longer HLA-DM-mediated half-lives.
Human cytotoxic CD4+ T cells recognize HLA-DR1-restricted epitopes on vaccinia virus proteins A24R and D1R conserved among poxviruses.
Human CD4+ T cell epitopes from vaccinia virus induced by vaccination or infection.
Bi-specific MHC heterodimers for characterization of cross-reactive T cells.
CD4+ T cells provide intermolecular help to generate robust antibody responses in vaccinia virus-vaccinated humans.
Disparate epitopes mediating protective heterologous immunity to unrelated viruses share peptide-MHC structural features recognized by cross-reactive T cells.