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Cantor, Sharon

One or more keywords matched the following items that are connected to Cantor, Sharon

Item Type	Name
Academic Article	The BRCA1-associated protein BACH1 is a DNA helicase targeted by clinically relevant inactivating mutations.
Academic Article	Analysis of the DNA substrate specificity of the human BACH1 helicase associated with breast cancer.
Academic Article	FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
Academic Article	Human MutL-complexes monitor homologous recombination independently of mismatch repair.
Academic Article	FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange.
Academic Article	Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repair.
Academic Article	An MLH1 mutation links BACH1/FANCJ to colon cancer, signaling, and insight toward directed therapy.
Academic Article	FANCJ/BACH1 acetylation at lysine 1249 regulates the DNA damage response.
Academic Article	BACH1 is critical for homologous recombination and appears to be the Fanconi anemia gene product FANCJ.
Academic Article	Assessing the link between BACH1 and BRCA1 in the FA pathway.
Academic Article	BACH1 is a DNA repair protein supporting BRCA1 damage response.
Academic Article	Inhibition of BACH1 (FANCJ) helicase by backbone discontinuity is overcome by increased motor ATPase or length of loading strand.
Academic Article	The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.
Academic Article	Targeting the FANCJ-BRCA1 interaction promotes a switch from recombination to poleta-dependent bypass.
Academic Article	Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome.
Academic Article	Hereditary breast cancer and the BRCA1-associated FANCJ/BACH1/BRIP1.
Academic Article	Fanconi anemia group J helicase and MRE11 nuclease interact to facilitate the DNA damage response.
Concept	Basic-Leucine Zipper Transcription Factors
Academic Article	FANCJ localization by mismatch repair is vital to maintain genomic integrity after UV irradiation.
Academic Article	Crosstalk between BRCA-Fanconi anemia and mismatch repair pathways prevents MSH2-dependent aberrant DNA damage responses.
Academic Article	FANCJ at the FORK.

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Basic Leucine Zipper Transcription Factors