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dSarm/Sarm1 is required for activation of an injury-induced axon death pathway.
DRK/DOS/SOS converge with Crk/Mbc/dCed-12 to activate Rac1 during glial engulfment of axonal debris.
Attenuated traumatic axonal injury and improved functional outcome after traumatic brain injury in mice lacking Sarm1.
Loss of Sarm1 does not suppress motor neuron degeneration in the SOD1G93A mouse model of amyotrophic lateral sclerosis.
Transcription factor Pebbled/RREB1 regulates injury-induced axon degeneration.
Axon Death Pathways Converge on Axundead to Promote Functional and Structural Axon Disassembly.
Injury-Induced Inhibition of Bystander Neurons Requires dSarm and Signaling from Glia.
TSG101 negatively regulates mitochondrial biogenesis in axons.
SARM1 signaling mechanisms in the injured nervous system.