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Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1.
Postprandial hepatic lipid metabolism requires signaling through Akt2 independent of the transcription factors FoxA2, FoxO1, and SREBP1c.
Rapamycin-induced insulin resistance is mediated by mTORC2 loss and uncoupled from longevity.
Adipocytes arise from multiple lineages that are heterogeneously and dynamically distributed.
Adipose tissue mTORC2 regulates ChREBP-driven de novo lipogenesis and hepatic glucose metabolism.
Systemic insulin sensitivity is regulated by GPS2 inhibition of AKT ubiquitination and activation in adipose tissue.
The Complex Roles of Mechanistic Target of Rapamycin in Adipocytes and Beyond.
Insulin PACS a Punch in SIRT1 Activity.
Adipocyte ACLY Facilitates Dietary Carbohydrate Handling to Maintain Metabolic Homeostasis in Females.
mTORC2/Akt activation in adipocytes is required for adipose tissue inflammation in tuberculosis.
De Novo Lipogenesis as a Source of Second Messengers in Adipocytes.
Proteome and Phosphoproteome Analysis of Brown Adipocytes Reveals That RICTOR Loss Dampens Global Insulin/AKT Signaling.
Integrating adipocyte insulin signaling and metabolism in the multi-omics era.
Proximity labeling of endogenous RICTOR identifies mTOR complex 2 regulation by ADP ribosylation factor ARF1.