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Guertin, David

One or more keywords matched the following items that are connected to Guertin, David

Item Type	Name
Academic Article	An expanding role for mTOR in cancer.
Academic Article	Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1.
Academic Article	Postprandial hepatic lipid metabolism requires signaling through Akt2 independent of the transcription factors FoxA2, FoxO1, and SREBP1c.
Academic Article	Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex.
Academic Article	Defining the role of mTOR in cancer.
Academic Article	mTOR complex 2 is required for the development of prostate cancer induced by Pten loss in mice.
Academic Article	Targeting mTOR: prospects for mTOR complex 2 inhibitors in cancer therapy.
Academic Article	Rapamycin-induced insulin resistance is mediated by mTORC2 loss and uncoupled from longevity.
Concept	Oncogenes
Concept	Proto-Oncogene Proteins c-akt
Concept	Proto-Oncogene Proteins
Academic Article	Rictor/mTORC2 loss in the Myf5 lineage reprograms brown fat metabolism and protects mice against obesity and metabolic disease.
Academic Article	Adipose tissue mTORC2 regulates ChREBP-driven de novo lipogenesis and hepatic glucose metabolism.
Academic Article	Systemic insulin sensitivity is regulated by GPS2 inhibition of AKT ubiquitination and activation in adipose tissue.
Academic Article	Brown Fat AKT2 Is a Cold-Induced Kinase that Stimulates ChREBP-Mediated De Novo Lipogenesis to Optimize Fuel Storage and Thermogenesis.
Academic Article	Oncogenic AKTivation by methylation.
Academic Article	Brown fat organogenesis and maintenance requires AKT1 and AKT2.
Academic Article	mTORC2/Akt activation in adipocytes is required for adipose tissue inflammation in tuberculosis.
Academic Article	Proteome and Phosphoproteome Analysis of Brown Adipocytes Reveals That RICTOR Loss Dampens Global Insulin/AKT Signaling.
Academic Article	mTORC2-AKT signaling to ATP-citrate lyase drives brown adipogenesis and de novo lipogenesis.

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Oncogenes