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Establishment of an in vivo meningioma model with human telomerase reverse transcriptase.
Natural history of meningioma development in mice reveals: a synergy of Nf2 and p16(Ink4a) mutations.
Genomic profiling reveals alternative genetic pathways of meningioma malignant progression dependent on the underlying NF2 status.
Alternative splicing of CHEK2 and codeletion with NF2 promote chromosomal instability in meningioma.