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MyD88-dependent activation of dendritic cells and CD4(+) T lymphocytes mediates symptoms, but is not required for the immunological control of parasites during rodent malaria.
Neutrophil paralysis in Plasmodium vivax malaria.
The CD14+CD16+ inflammatory monocyte subset displays increased mitochondrial activity and effector function during acute Plasmodium vivax malaria.
Induction of Inhibitory Receptors on T Cells During Plasmodium vivax Malaria Impairs Cytokine Production.
Type I Interferon Transcriptional Signature in Neutrophils and Low-Density Granulocytes Are Associated with Tissue Damage in Malaria.
T follicular helper cells regulate the activation of B lymphocytes and antibody production during Plasmodium vivax infection.
Plasmodium vivax Infection Impairs Regulatory T-Cell Suppressive Function During Acute Malaria.
The emergence of pathogenic TNF/iNOS producing dendritic cells (Tip-DCs) in a malaria model of acute respiratory distress syndrome (ARDS) is dependent on CCR4.
Cytotoxic CD8+ T cells recognize and kill Plasmodium vivax-infected reticulocytes.
Insights into CD4+ memory T cells following Leishmania infection.
Monocyte-derived dendritic cells in malaria.
The immunology of Plasmodium vivax malaria.
Plasmodium vivax Infection Alters Mitochondrial Metabolism in Human Monocytes.
Asymptomatic Plasmodium vivax malaria in the Brazilian Amazon: Submicroscopic parasitemic blood infects Nyssorhynchus darlingi.