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Resistance of HIV-infected macrophages to CD8+ T lymphocyte-mediated killing drives activation of the immune system.
Tim-3 negatively regulates cytotoxicity in exhausted CD8+ T cells in HIV infection.
TIGIT Marks Exhausted T Cells, Correlates with Disease Progression, and Serves as a Target for Immune Restoration in HIV and SIV Infection.
HIV-infected macrophages resist efficient NK cell-mediated killing while preserving inflammatory cytokine responses.
Antigen-independent induction of Tim-3 expression on human T cells by the common ?-chain cytokines IL-2, IL-7, IL-15, and IL-21 is associated with proliferation and is dependent on the phosphoinositide 3-kinase pathway.
HERV-K-specific T cells eliminate diverse HIV-1/2 and SIV primary isolates.
CD4 Positive T Lymphocytes