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Clayton, Kiera
Item Type | Name |
Concept
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HIV Infections
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Academic Article
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Resistance of HIV-infected macrophages to CD8+ T lymphocyte-mediated killing drives activation of the immune system.
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Academic Article
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Tim-3 negatively regulates cytotoxicity in exhausted CD8+ T cells in HIV infection.
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Academic Article
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HIV Infection of Macrophages: Implications for Pathogenesis and Cure.
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Academic Article
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TIGIT Marks Exhausted T Cells, Correlates with Disease Progression, and Serves as a Target for Immune Restoration in HIV and SIV Infection.
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Academic Article
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Tim-3 is a Marker of Plasmacytoid Dendritic Cell Dysfunction during HIV Infection and Is Associated with the Recruitment of IRF7 and p85 into Lysosomes and with the Submembrane Displacement of TLR9.
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Academic Article
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HIV-infected macrophages resist efficient NK cell-mediated killing while preserving inflammatory cytokine responses.
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Academic Article
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T cell Ig and mucin domain-containing protein 3 is recruited to the immune synapse, disrupts stable synapse formation, and associates with receptor phosphatases.
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Academic Article
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HCV-specific T cells in HCV/HIV co-infection show elevated frequencies of dual Tim-3/PD-1 expression that correlate with liver disease progression.
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Academic Article
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IL-10-producing B cells are induced early in HIV-1 infection and suppress HIV-1-specific T cell responses.
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Academic Article
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Antigen-independent induction of Tim-3 expression on human T cells by the common ?-chain cytokines IL-2, IL-7, IL-15, and IL-21 is associated with proliferation and is dependent on the phosphoinositide 3-kinase pathway.
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Academic Article
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HERV-K-specific T cells eliminate diverse HIV-1/2 and SIV primary isolates.
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Academic Article
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Soluble T cell immunoglobulin mucin domain 3 is shed from CD8+ T cells by the sheddase ADAM10, is increased in plasma during untreated HIV infection, and correlates with HIV disease progression.
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Academic Article
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Trimeric HIV-1 gp140 fused with APRIL, BAFF, and CD40L on the mucosal gp140-specific antibody responses in mice.
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Academic Article
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Harnessing immune cells to eliminate HIV reservoirs.
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Academic Article
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HIV-1 latency reversal agent boosting is not limited by opioid use.
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