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Item TypeName
Concept HIV Infections
Academic Article Resistance of HIV-infected macrophages to CD8+ T lymphocyte-mediated killing drives activation of the immune system.
Academic Article Tim-3 negatively regulates cytotoxicity in exhausted CD8+ T cells in HIV infection.
Academic Article HIV Infection of Macrophages: Implications for Pathogenesis and Cure.
Academic Article TIGIT Marks Exhausted T Cells, Correlates with Disease Progression, and Serves as a Target for Immune Restoration in HIV and SIV Infection.
Academic Article Tim-3 is a Marker of Plasmacytoid Dendritic Cell Dysfunction during HIV Infection and Is Associated with the Recruitment of IRF7 and p85 into Lysosomes and with the Submembrane Displacement of TLR9.
Academic Article HIV-infected macrophages resist efficient NK cell-mediated killing while preserving inflammatory cytokine responses.
Academic Article T cell Ig and mucin domain-containing protein 3 is recruited to the immune synapse, disrupts stable synapse formation, and associates with receptor phosphatases.
Academic Article HCV-specific T cells in HCV/HIV co-infection show elevated frequencies of dual Tim-3/PD-1 expression that correlate with liver disease progression.
Academic Article IL-10-producing B cells are induced early in HIV-1 infection and suppress HIV-1-specific T cell responses.
Academic Article Antigen-independent induction of Tim-3 expression on human T cells by the common ?-chain cytokines IL-2, IL-7, IL-15, and IL-21 is associated with proliferation and is dependent on the phosphoinositide 3-kinase pathway.
Academic Article HERV-K-specific T cells eliminate diverse HIV-1/2 and SIV primary isolates.
Academic Article Soluble T cell immunoglobulin mucin domain 3 is shed from CD8+ T cells by the sheddase ADAM10, is increased in plasma during untreated HIV infection, and correlates with HIV disease progression.
Academic Article Trimeric HIV-1 gp140 fused with APRIL, BAFF, and CD40L on the mucosal gp140-specific antibody responses in mice.
Academic Article Harnessing immune cells to eliminate HIV reservoirs.
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  • HIV Infections