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One or more keywords matched the following items that are connected to Quinton, Lee
Item TypeName
Concept STAT3 Transcription Factor
Concept STAT1 Transcription Factor
Concept Transcription Factor RelA
Academic Article Induction of STAT3-Dependent CXCL5 Expression and Neutrophil Recruitment by Oncostatin-M during Pneumonia.
Academic Article Roles of STAT3 in protein secretion pathways during the acute-phase response.
Academic Article Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.
Academic Article NF-?B RelA Is Required for Hepatoprotection during Pneumonia and Sepsis.
Academic Article Alveolar epithelial STAT3, IL-6 family cytokines, and host defense during Escherichia coli pneumonia.
Academic Article The granulopoietic cytokine response and enhancement of granulopoiesis in mice during endotoxemia.
Academic Article Roles of interleukin-6 in activation of STAT proteins and recruitment of neutrophils during Escherichia coli pneumonia.
Academic Article Mechanisms of the hepatic acute-phase response during bacterial pneumonia.
Academic Article NF-?B and STAT3 signaling hubs for lung innate immunity.
Academic Article Myeloid-epithelial cross talk coordinates synthesis of the tissue-protective cytokine leukemia inhibitory factor during pneumonia.
Academic Article Acute alcohol intoxication suppresses the pulmonary ELR-negative CXC chemokine response to lipopolysaccharide.
Academic Article Leukemia inhibitory factor signaling is required for lung protection during pneumonia.
Academic Article Functions and regulation of NF-kappaB RelA during pneumococcal pneumonia.
Academic Article Hepatocyte-specific mutation of both NF-?B RelA and STAT3 abrogates the acute phase response in mice.
Academic Article Type I alveolar epithelial cells mount innate immune responses during pneumococcal pneumonia.
Academic Article Activation of Hepatic STAT3 Maintains Pulmonary Defense during Endotoxemia.
Academic Article Liver-Dependent Lung Remodeling during Systemic Inflammation Shapes Responses to Secondary Infection.
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  • Transcription Factors