"Pulmonary Eosinophilia" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A condition characterized by infiltration of the lung with EOSINOPHILS due to inflammation or other disease processes. Major eosinophilic lung diseases are the eosinophilic pneumonias caused by infections, allergens, or toxic agents.
| Descriptor ID |
D011657
|
| MeSH Number(s) |
C08.381.750 C15.378.553.231.549.750
|
| Concept/Terms |
Pulmonary Eosinophilia- Pulmonary Eosinophilia
- Eosinophilias, Pulmonary
- Pulmonary Eosinophilias
- Eosinophilia, Pulmonary
Simple Pulmonary Eosinophilia- Simple Pulmonary Eosinophilia
- Pulmonary Eosinophilia, Simple
- Simple Pulmonary Eosinophilias
- Loeffler Syndrome
- Syndrome, Loeffler
Tropical Eosinophilic Pneumonia- Tropical Eosinophilic Pneumonia
- Eosinophilic Pneumonia, Tropical
- Eosinophilic Pneumonias, Tropical
- Tropical Eosinophilic Pneumonias
Pneumonia, Eosinophilic- Pneumonia, Eosinophilic
- Eosinophilic Pneumonias
- Pneumonias, Eosinophilic
- Eosinophilic Pneumonia
|
Below are MeSH descriptors whose meaning is more general than "Pulmonary Eosinophilia".
Below are MeSH descriptors whose meaning is more specific than "Pulmonary Eosinophilia".
This graph shows the total number of publications written about "Pulmonary Eosinophilia" by people in this website by year, and whether "Pulmonary Eosinophilia" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 1 | 1 | 2 |
| 2001 | 1 | 1 | 2 |
| 2015 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
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click here.
Below are the most recent publications written about "Pulmonary Eosinophilia" by people in Profiles.
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Murphy DJ, Sabir R, Agarwal KA, Alroumi F. Acute Eosinophilic Pneumonia Presenting with an Elevated Procalcitonin Level: A Rare Laboratory Finding. Am J Case Rep. 2019 Nov 28; 20:1765-1768.
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Mart?nez-Balzano CD, Sosa A, O'Reilly S, Gong Y, Irwin R. A 66-Year-Old Man With a Past History of Relapsing Polychondritis Presented With Right Upper Lobe Consolidation, Nodular Airway Lesions, and a Corticosteroid-Responsive Chronic Cough. Chest. 2015 Nov; 148(5):e142-e147.
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Verhoef PA, Constantinides MG, McDonald BD, Urban JF, Sperling AI, Bendelac A. Intrinsic functional defects of type 2 innate lymphoid cells impair innate allergic inflammation in promyelocytic leukemia zinc finger (PLZF)-deficient mice. J Allergy Clin Immunol. 2016 Feb; 137(2):591-600.e1.
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McConchie BW, Norris HH, Bundoc VG, Trivedi S, Boesen A, Urban JF, Keane-Myers AM. Ascaris suum-derived products suppress mucosal allergic inflammation in an interleukin-10-independent manner via interference with dendritic cell function. Infect Immun. 2006 Dec; 74(12):6632-41.
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Mathew A, Medoff BD, Carafone AD, Luster AD. Cutting edge: Th2 cell trafficking into the allergic lung is dependent on chemoattractant receptor signaling. J Immunol. 2002 Jul 15; 169(2):651-5.
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Lilly CM, Daugherty BL. A novel LPS-inducible CCR3 activator: why so many CCR3 ligands? Am J Respir Cell Mol Biol. 2001 Dec; 25(6):673-5.
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Tateno H, Nakamura H, Minematsu N, Amakawa K, Terashima T, Fujishima S, Luster AD, Lilly CM, Yamaguchi K. Eotaxin and monocyte chemoattractant protein-1 in chronic eosinophilic pneumonia. Eur Respir J. 2001 May; 17(5):962-8.
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Schwarze J, Cieslewicz G, Joetham A, Ikemura T, M?kel? MJ, Dakhama A, Shultz LD, Lamers MC, Gelfand EW. Critical roles for interleukin-4 and interleukin-5 during respiratory syncytial virus infection in the development of airway hyperresponsiveness after airway sensitization. Am J Respir Crit Care Med. 2000 Aug; 162(2 Pt 1):380-6.
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Krieg AM, Kline JN. Immune effects and therapeutic applications of CpG motifs in bacterial DNA. Immunopharmacology. 2000 Jul 25; 48(3):303-5.
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O'Sullivan BP, Nimkin K, Gang DL. A fifteen-year-old boy with eosinophilia and pulmonary infiltrates. J Pediatr. 1993 Oct; 123(4):660-6.