"Bacteriophage phi X 174" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The type species of the genus MICROVIRUS. A prototype of the small virulent DNA coliphages, it is composed of a single strand of supercoiled circular DNA, which on infection, is converted to a double-stranded replicative form by a host enzyme.
Descriptor ID |
D010584
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MeSH Number(s) |
B04.123.205.320 B04.123.470.500.320 B04.280.470.500.320
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Concept/Terms |
Bacteriophage phi X 174- Bacteriophage phi X 174
- Enterobacteria phage phi X 174
- phi X 174 Phage
- Phage phi X174
- Coliphage phi X 174
- Phage phi X 174
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Below are MeSH descriptors whose meaning is more general than "Bacteriophage phi X 174".
Below are MeSH descriptors whose meaning is more specific than "Bacteriophage phi X 174".
This graph shows the total number of publications written about "Bacteriophage phi X 174" by people in this website by year, and whether "Bacteriophage phi X 174" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1996 | 0 | 1 | 1 |
1999 | 1 | 0 | 1 |
2005 | 0 | 1 | 1 |
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Below are the most recent publications written about "Bacteriophage phi X 174" by people in Profiles.
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Calmann MA, Evans JE, Marinus MG. MutS inhibits RecA-mediated strand transfer with methylated DNA substrates. Nucleic Acids Res. 2005; 33(11):3591-7.
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Repik A, Pincus SE, Ghiran I, Nicholson-Weller A, Asher DR, Cerny AM, Casey LS, Jones SM, Jones SN, Mohamed N, Klickstein LB, Spitalny G, Finberg RW. A transgenic mouse model for studying the clearance of blood-borne pathogens via human complement receptor 1 (CR1). Clin Exp Immunol. 2005 May; 140(2):230-40.
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Liu J, Xu L, Sandler SJ, Marians KJ. Replication fork assembly at recombination intermediates is required for bacterial growth. Proc Natl Acad Sci U S A. 1999 Mar 30; 96(7):3552-5.
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Sandler SJ, Samra HS, Clark AJ. Differential suppression of priA2::kan phenotypes in Escherichia coli K-12 by mutations in priA, lexA, and dnaC. Genetics. 1996 May; 143(1):5-13.