N-Formylmethionine Leucyl-Phenylalanine
"N-Formylmethionine Leucyl-Phenylalanine" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A formylated tripeptide originally isolated from bacterial filtrates that is positively chemotactic to polymorphonuclear leucocytes, and causes them to release lysosomal enzymes and become metabolically activated.
| Descriptor ID |
D009240
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| MeSH Number(s) |
D02.886.030.676.450.440 D12.125.072.050.685.445 D12.125.142.666.500 D12.125.166.676.450.440 D12.644.456.400 D23.125.685
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| Concept/Terms |
N-Formylmethionine Leucyl-Phenylalanine- N-Formylmethionine Leucyl-Phenylalanine
- Leucyl-Phenylalanine, N-Formylmethionine
- N Formylmethionine Leucyl Phenylalanine
- Formylmethionyl-Leucyl-Phenylalanine
- Formylmethionyl Leucyl Phenylalanine
- N-Formyl-Methionyl-Leucyl-Phenylalanine
- N Formyl Methionyl Leucyl Phenylalanine
- Formylmet-Leu-Phe
- Formylmet Leu Phe
- Formylmethionylleucylphenylalanine
Formylmethionyl Peptide- Formylmethionyl Peptide
- Peptide, Formylmethionyl
- N-Formylated Peptide
- N Formylated Peptide
- Peptide, N-Formylated
|
Below are MeSH descriptors whose meaning is more general than "N-Formylmethionine Leucyl-Phenylalanine".
Below are MeSH descriptors whose meaning is more specific than "N-Formylmethionine Leucyl-Phenylalanine".
This graph shows the total number of publications written about "N-Formylmethionine Leucyl-Phenylalanine" by people in this website by year, and whether "N-Formylmethionine Leucyl-Phenylalanine" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 1999 | 0 | 1 | 1 |
| 2003 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
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Below are the most recent publications written about "N-Formylmethionine Leucyl-Phenylalanine" by people in Profiles.
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Taylor A, Tang W, Bruscia EM, Zhang PX, Lin A, Gaines P, Wu D, Halene S. SRF is required for neutrophil migration in response to inflammation. Blood. 2014 May 08; 123(19):3027-36.
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Zhang X, Kluger Y, Nakayama Y, Poddar R, Whitney C, DeTora A, Weissman SM, Newburger PE. Gene expression in mature neutrophils: early responses to inflammatory stimuli. J Leukoc Biol. 2004 Feb; 75(2):358-72.
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McCormick BA, Fernandez MI, Siber AM, Maurelli AT. Inhibition of Shigella flexneri-induced transepithelial migration of polymorphonuclear leucocytes by cadaverine. Cell Microbiol. 1999 Sep; 1(2):143-55.
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Shaw SK, Hermanowski-Vosatka A, Shibahara T, McCormick BA, Parkos CA, Carlson SL, Ebert EC, Brenner MB, Madara JL. Migration of intestinal intraepithelial lymphocytes into a polarized epithelial monolayer. Am J Physiol. 1998 09; 275(3):G584-91.
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Bernardo J, Brennan L, Brink HF, Ortiz MF, Newburger PE, Simons ER. Chemotactic peptide-induced cytoplasmic pH changes in incubated human monocytes. J Leukoc Biol. 1993 Jun; 53(6):673-8.
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Lynn WA, Raetz CR, Qureshi N, Golenbock DT. Lipopolysaccharide-induced stimulation of CD11b/CD18 expression on neutrophils. Evidence of specific receptor-based response and inhibition by lipid A-based antagonists. J Immunol. 1991 Nov 01; 147(9):3072-9.
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Bernardo J, Newburger PE, Brennan L, Brink HF, Bresnick SA, Weil G, Simons ER. Simultaneous flow cytometric measurements of cytoplasmic Ca++ and membrane potential changes upon FMLP exposure as HL-60 cells mature into granulocytes: using [Ca++]in as an indicator of granulocyte maturity. J Leukoc Biol. 1990 Mar; 47(3):265-74.
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Simpkins CO, Mazorow DL, Alailima ST, Tate EA, Sweatt W, Johnson M, Shariff K, Millar DB. Prostaglandin D2 modulates human neutrophil intracellular calcium flux and inhibits superoxide release via its ring carbonyl. Life Sci. 1990; 46(11):793-801.
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Tauber AI, Pavlotsky N, Lin JS, Rice PA. Inhibition of human neutrophil NADPH oxidase by Chlamydia serovars E, K, and L2. Infect Immun. 1989 Apr; 57(4):1108-12.
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Georgilis K, Klempner MS. In vitro effects of omega-3 fatty acids on neutrophil intracellular calcium homeostasis and receptor expression for FMLP and LTB4. Inflammation. 1988 Oct; 12(5):475-90.