Glucose Intolerance

"Glucose Intolerance" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

A pathological state in which BLOOD GLUCOSE level is less than approximately 140 mg/100 ml of PLASMA at fasting, and above approximately 200 mg/100 ml plasma at 30-, 60-, or 90-minute during a GLUCOSE TOLERANCE TEST. This condition is seen frequently in DIABETES MELLITUS, but also occurs with other diseases and MALNUTRITION.

Descriptor ID	D018149
MeSH Number(s)	C18.452.394.952.500
Concept/Terms	Glucose Intolerance Glucose Intolerance Glucose Intolerances Intolerance, Glucose Intolerances, Glucose Impaired Glucose Tolerance Glucose Tolerance, Impaired Glucose Tolerances, Impaired Impaired Glucose Tolerances Tolerance, Impaired Glucose Tolerances, Impaired Glucose

Below are MeSH descriptors whose meaning is more general than "Glucose Intolerance".

Diseases [C]
Nutritional and Metabolic Diseases [C18]
Metabolic Diseases [C18.452]
Glucose Metabolism Disorders [C18.452.394]
Hyperglycemia [C18.452.394.952]
Glucose Intolerance [C18.452.394.952.500]

Below are MeSH descriptors whose meaning is related to "Glucose Intolerance".

Below are MeSH descriptors whose meaning is more specific than "Glucose Intolerance".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Glucose Intolerance" by people in this website by year, and whether "Glucose Intolerance" was a major or minor topic of these publications.

Bar chart showing 53 publications over 19 distinct years, with a maximum of 7 publications in 2013

To see the data from this visualization as text, click here.

Year	Major Topic	Minor Topic	Total
1996	0	1	1
2002	1	0	1
2003	1	1	2
2004	1	0	1
2006	2	0	2
2007	0	1	1
2008	1	1	2
2009	1	1	2
2010	2	0	2
2011	0	3	3
2012	2	2	4
2013	7	0	7
2014	2	2	4
2015	4	1	5
2016	3	3	6
2017	2	1	3
2018	0	1	1
2020	2	0	2
2021	1	3	4

Below are the most recent publications written about "Glucose Intolerance" by people in Profiles.

Tsagkaraki E, Nicoloro SM, DeSouza T, Solivan-Rivera J, Desai A, Lifshitz LM, Shen Y, Kelly M, Guilherme A, Henriques F, Amrani N, Ibraheim R, Rodriguez TC, Luk K, Maitland S, Friedline RH, Tauer L, Hu X, Kim JK, Wolfe SA, Sontheimer EJ, Corvera S, Czech MP. CRISPR-enhanced human adipocyte browning as cell therapy for metabolic disease. Nat Commun. 2021 11 26; 12(1):6931.

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Mills EL, Harmon C, Jedrychowski MP, Xiao H, Garrity R, Tran NV, Bradshaw GA, Fu A, Szpyt J, Reddy A, Prendeville H, Danial NN, Gygi SP, Lynch L, Chouchani ET. UCP1 governs liver extracellular succinate and inflammatory pathogenesis. Nat Metab. 2021 05; 3(5):604-617.

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Darukhanavala A, Van Dessel F, Ho J, Hansen M, Kremer T, Alfego D. Use of hemoglobin A1c to identify dysglycemia in cystic fibrosis. PLoS One. 2021; 16(4):e0250036.

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Fujimoto BA, Young M, Nakamura N, Ha H, Carter L, Pitts MW, Torres D, Noh HL, Suk S, Kim JK, Polgar N. Disrupted glucose homeostasis and skeletal-muscle-specific glucose uptake in an exocyst knockout mouse model. J Biol Chem. 2021 Jan-Jun; 296:100482.

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Wang G, Yu Y, Cai W, Batista TM, Suk S, Noh HL, Hirshman M, Nigro P, Li ME, Softic S, Goodyear L, Kim JK, Kahn CR. Muscle-Specific Insulin Receptor Overexpression Protects Mice From Diet-Induced Glucose Intolerance but Leads to Postreceptor Insulin Resistance. Diabetes. 2020 11; 69(11):2294-2309.

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van der Vinne V, Martin Burgos B, Harrington ME, Weaver DR. Deconstructing circadian disruption: Assessing the contribution of reduced peripheral oscillator amplitude on obesity and glucose intolerance in mice. J Pineal Res. 2020 Aug; 69(1):e12654.

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Craige SM, Chen K, Blanton RM, Keaney JF, Kant S. JNK and cardiometabolic dysfunction. Biosci Rep. 2019 07 31; 39(7).

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Huang LH, Melton EM, Li H, Sohn P, Jung D, Tsai CY, Ma T, Sano H, Ha H, Friedline RH, Kim JK, Usherwood E, Chang CCY, Chang TY. Myeloid-specific Acat1 ablation attenuates inflammatory responses in macrophages, improves insulin sensitivity, and suppresses diet-induced obesity. Am J Physiol Endocrinol Metab. 2018 09 01; 315(3):E340-E356.

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Huang L, Pan D, Chen Q, Zhu LJ, Ou J, Wabitsch M, Wang YX. Transcription factor Hlx controls a systematic switch from white to brown fat through Prdm16-mediated co-activation. Nat Commun. 2017 07 12; 8(1):68.

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Zhang X, Devlin HM, Smith B, Imperatore G, Thomas W, Lobelo F, Ali MK, Norris K, Gruss S, Bardenheier B, Cho P, Garcia de Quevedo I, Mudaliar U, Jones CD, Durthaler JM, Saaddine J, Geiss LS, Gregg EW. Effect of lifestyle interventions on cardiovascular risk factors among adults without impaired glucose tolerance or diabetes: A systematic review and meta-analysis. PLoS One. 2017; 12(5):e0176436.

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