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The Drosophila Toll-9 activates a constitutive antimicrobial defense.
Hopelessness and complementary therapy use in patients with ovarian cancer.
Toll and Toll-9 in Drosophila innate immune response.
HIV-1 Vpr induces defects in mitosis, cytokinesis, nuclear structure, and centrosomes.
Disruption of PDGF receptor trafficking by mutation of its PI-3 kinase binding sites.
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Disruption of PDGF receptor trafficking by mutation of its PI-3 kinase binding sites.
Joly M, Kazlauskas A, Fay FS, Corvera S. Disruption of PDGF receptor trafficking by mutation of its PI-3 kinase binding sites. Science. 1994 Feb 04; 263(5147):684-7.
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subject areas
Binding Sites
Cell Membrane
Endocytosis
Golgi Apparatus
GTPase-Activating Proteins
Humans
Intracellular Signaling Peptides and Proteins
Isoenzymes
Mutation
Phosphatidylinositol 3-Kinases
Phospholipase C gamma
Phosphotransferases (Alcohol Group Acceptor)
Platelet-Derived Growth Factor
Protein Tyrosine Phosphatase, Non-Receptor Type 11
Protein Tyrosine Phosphatase, Non-Receptor Type 6
Protein Tyrosine Phosphatases
Proteins
ras GTPase-Activating Proteins
Receptors, Platelet-Derived Growth Factor
Tumor Cells, Cultured
Type C Phospholipases
authors with profiles
Silvia Corvera MD