"Prostaglandins E" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
(11 alpha,13E,15S)-11,15-Dihydroxy-9-oxoprost-13-en-1-oic acid (PGE(1)); (5Z,11 alpha,13E,15S)-11,15-dihydroxy-9-oxoprosta-5,13-dien-1-oic acid (PGE(2)); and (5Z,11 alpha,13E,15S,17Z)-11,15-dihydroxy-9-oxoprosta-5,13,17-trien-1-oic acid (PGE(3)). Three of the six naturally occurring prostaglandins. They are considered primary in that no one is derived from another in living organisms. Originally isolated from sheep seminal fluid and vesicles, they are found in many organs and tissues and play a major role in mediating various physiological activities.
| Descriptor ID |
D011458
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| MeSH Number(s) |
D10.251.355.255.550.250 D23.469.050.175.725.250
|
| Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Prostaglandins E".
Below are MeSH descriptors whose meaning is more specific than "Prostaglandins E".
This graph shows the total number of publications written about "Prostaglandins E" by people in this website by year, and whether "Prostaglandins E" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1993 | 0 | 1 | 1 |
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Below are the most recent publications written about "Prostaglandins E" by people in Profiles.
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Kitagawa Y, Desemone J, Mordes JP. Pancreas-specific venular labeling by monastral blue B in the BB rat: modulation by prostaglandins and their inhibitors. Immunopharmacology. 1993 May-Jun; 25(3):229-38.
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Callegari PE, Zurier RB. Botanical lipids: potential role in modulation of immunologic responses and inflammatory reactions. Rheum Dis Clin North Am. 1991 May; 17(2):415-25.
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Santoli D, Phillips PD, Colt TL, Zurier RB. Suppression of interleukin 2-dependent human T cell growth in vitro by prostaglandin E (PGE) and their precursor fatty acids. Evidence for a PGE-independent mechanism of inhibition by the fatty acids. J Clin Invest. 1990 Feb; 85(2):424-32.
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Baker DG, Krakauer KA, Tate G, Laposata M, Zurier RB. Suppression of human synovial cell proliferation by dihomo-gamma-linolenic acid. Arthritis Rheum. 1989 Oct; 32(10):1273-81.
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Styrt B, Klempner MS, Rocklin RE. Comparison of prostaglandins E2 and D2 as inhibitors of respiratory burst in neutrophils from atopic and nonatopic subjects. Inflammation. 1988 Jun; 12(3):213-21.
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Brezis M, Rosen S, Stoff JS, Spokes K, Silva P, Epstein FH. Inhibition of prostaglandin synthesis in rat kidney perfused with and without erythrocytes: implication for analgesic nephropathy. Miner Electrolyte Metab. 1986; 12(5-6):326-32.
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Whittum J, Goldschneider I, Greiner D, Zurier R. Developmental abnormalities of terminal deoxynucleotidyl transferase positive bone marrow cells and thymocytes in New Zealand mice: effects of prostaglandin E1. J Immunol. 1985 Jul; 135(1):272-80.
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Morain WD, Pettit RJ, Rothkopf DM, Coombs DW. Augmentation of surviving flap area by intraarterial vasodilators administered through implantable pumps. Ann Plast Surg. 1983 Jul; 11(1):46-52.
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Musch MW, Kachur JF, Miller RJ, Field M, Stoff JS. Bradykinin-stimulated electrolyte secretion in rabbit and guinea pig intestine. Involvement of arachidonic acid metabolites. J Clin Invest. 1983 May; 71(5):1073-83.
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Klein LA, Stoff JS. Prostaglandins and the prostate: an hypothesis on the etiology of benign prostatic hyperplasia. Prostate. 1983; 4(3):247-51.