 Membrane Potential, Mitochondrial
"Membrane Potential, Mitochondrial" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The voltage difference, normally maintained at approximately -180mV, across the INNER MITOCHONDRIAL MEMBRANE, by a net movement of positive charge across the membrane. It is a major component of the PROTON MOTIVE FORCE in MITOCHONDRIA used to drive the synthesis of ATP.
| Descriptor ID |
D053078
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| MeSH Number(s) |
G03.495.335.770.500 G04.580.550 G07.265.750.550
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| Concept/Terms |
Membrane Potential, Mitochondrial- Membrane Potential, Mitochondrial
- Membrane Potentials, Mitochondrial
- Mitochondrial Membrane Potentials
- Potential, Mitochondrial Membrane
- Potentials, Mitochondrial Membrane
- Mitochondrial Membrane Potential
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Below are MeSH descriptors whose meaning is more general than "Membrane Potential, Mitochondrial".
Below are MeSH descriptors whose meaning is more specific than "Membrane Potential, Mitochondrial".
This graph shows the total number of publications written about "Membrane Potential, Mitochondrial" by people in this website by year, and whether "Membrane Potential, Mitochondrial" was a major or minor topic of these publications.
To see the data from this visualization as text, click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 1 | 3 | 4 | | 2007 | 0 | 3 | 3 | | 2008 | 0 | 4 | 4 | | 2009 | 0 | 4 | 4 | | 2010 | 0 | 3 | 3 | | 2011 | 0 | 4 | 4 | | 2012 | 1 | 3 | 4 | | 2013 | 0 | 7 | 7 | | 2014 | 0 | 3 | 3 | | 2015 | 2 | 2 | 4 | | 2017 | 0 | 1 | 1 |
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Below are the most recent publications written about "Membrane Potential, Mitochondrial" by people in Profiles.
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Teixeira J, Cagide F, Benfeito S, Soares P, Garrido J, Baldeiras I, Ribeiro JA, Pereira CM, Silva AF, Andrade PB, Oliveira PJ, Borges F. Development of a Mitochondriotropic Antioxidant Based on Caffeic Acid: Proof of Concept on Cellular and Mitochondrial Oxidative Stress Models. J Med Chem. 2017 Aug 24; 60(16):7084-7098.
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Gonçalves IO, Passos E, Diogo CV, Rocha-Rodrigues S, Santos-Alves E, Oliveira PJ, Ascensão A, Magalhães J. Exercise mitigates mitochondrial permeability transition pore and quality control mechanisms alterations in nonalcoholic steatohepatitis. Appl Physiol Nutr Metab. 2016 Mar; 41(3):298-306.
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Zhao W, Jiang G, Bi C, Li Y, Liu J, Ye C, He H, Li L, Song D, Shao R. The dual topoisomerase inhibitor A35 preferentially and specially targets topoisomerase 2a by enhancing pre-strand and post-strand cleavage and inhibiting DNA religation. Oncotarget. 2015 Nov 10; 6(35):37871-94.
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Moreno AJ, Santos DL, Magalhães-Novais S, Oliveira PJ. Measuring Mitochondrial Membrane Potential with a Tetraphenylphosphonium-Selective Electrode. Curr Protoc Toxicol. 2015 Aug 06; 65:25.5.1-16.
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Loureiro R, Magalhães-Novais S, Mesquita KA, Baldeiras I, Sousa IS, Tavares LC, Barbosa IA, Oliveira PJ, Vega-Naredo I. Melatonin antiproliferative effects require active mitochondrial function in embryonal carcinoma cells. Oncotarget. 2015 Jul 10; 6(19):17081-96.
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Serafim TL, Carvalho FS, Bernardo TC, Pereira GC, Perkins E, Holy J, Krasutsky DA, Kolomitsyna ON, Krasutsky PA, Oliveira PJ. New derivatives of lupane triterpenoids disturb breast cancer mitochondria and induce cell death. Bioorg Med Chem. 2014 Nov 1; 22(21):6270-87.
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Deus CM, Coelho AR, Serafim TL, Oliveira PJ. Targeting mitochondrial function for the treatment of breast cancer. Future Med Chem. 2014 Sep; 6(13):1499-513.
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Yang ZF, Drumea K, Mott S, Wang J, Rosmarin AG. GABP transcription factor (nuclear respiratory factor 2) is required for mitochondrial biogenesis. Mol Cell Biol. 2014 Sep; 34(17):3194-201.
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Abdul-Sater AA, Tattoli I, Jin L, Grajkowski A, Levi A, Koller BH, Allen IC, Beaucage SL, Fitzgerald KA, Ting JP, Cambier JC, Girardin SE, Schindler C. Cyclic-di-GMP and cyclic-di-AMP activate the NLRP3 inflammasome. EMBO Rep. 2013 Oct; 14(10):900-6.
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Branco AF, Sampaio SF, Wieckowski MR, Sardão VA, Oliveira PJ. Mitochondrial disruption occurs downstream from ß-adrenergic overactivation by isoproterenol in differentiated, but not undifferentiated H9c2 cardiomyoblasts: differential activation of stress and survival pathways. Int J Biochem Cell Biol. 2013 Nov; 45(11):2379-91.
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