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A network of high-mobility group box transcription factors programs innate interleukin-17 production.
beta2 microglobulin-deficient (B2m(null)) NOD/SCID mice are excellent recipients for studying human stem cell function.
Brief physician and nurse practitioner-delivered counseling for high-risk drinking. Results at 12-month follow-up.
Rapid destruction of encapsulated islet xenografts by NOD mice is CD4-dependent and facilitated by B-cells: innate immunity and autoimmunity do not play significant roles.
Invasion of the killer B's in type 1 diabetes.
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Invasion of the killer B's in type 1 diabetes.
Silveira PA, Serreze DV, Grey ST. Invasion of the killer B's in type 1 diabetes. Front Biosci. 2007 Jan 01; 12:2183-93.
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PubMed
subject areas
Animals
Antigen-Presenting Cells
B-Lymphocyte Subsets
B-Lymphocytes
CD4-Positive T-Lymphocytes
Diabetes Mellitus, Type 1
H-2 Antigens
Humans
Mice
Mice, Inbred NOD
Self Tolerance
authors with profiles
David V Serreze PhD