Epistasis, Genetic

"Epistasis, Genetic" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

A form of gene interaction whereby the expression of one gene interferes with or masks the expression of a different gene or genes. Genes whose expression interferes with or masks the effects of other genes are said to be epistatic to the effected genes. Genes whose expression is affected (blocked or masked) are hypostatic to the interfering genes.

Descriptor ID	D004843
MeSH Number(s)	G05.308.207
Concept/Terms	Epistasis, Genetic Epistasis, Genetic Epistases, Genetic Gene-Gene Interactions, Epistatic Gene Gene Interactions, Epistatic Gene-Gene Interaction, Epistatic Epistatic Gene-Gene Interaction Epistatic Gene-Gene Interactions Gene Gene Interaction, Epistatic Interaction, Epistatic Gene-Gene Interactions, Epistatic Gene-Gene Non-Allelic Gene Interactions Gene Interaction, Non-Allelic Gene Interactions, Non-Allelic Interaction, Non-Allelic Gene Interactions, Non-Allelic Gene Non Allelic Gene Interactions Non-Allelic Gene Interaction Genetic Epistasis Genetic Epistases Genes, Epistatic Genes, Epistatic Epistatic Gene Epistatic Genes Gene, Epistatic Genes, Hypostatic Genes, Hypostatic Gene, Hypostatic Hypostatic Gene Hypostatic Genes Deviation, Epistatic Deviation, Epistatic Interaction Deviation Epistatic Deviation

Below are MeSH descriptors whose meaning is more general than "Epistasis, Genetic".

Biological Sciences [G]
Genetic Phenomena [G05]
Gene Expression Regulation [G05.308]
Epistasis, Genetic [G05.308.207]

Below are MeSH descriptors whose meaning is more specific than "Epistasis, Genetic".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Epistasis, Genetic" by people in this website by year, and whether "Epistasis, Genetic" was a major or minor topic of these publications.

Bar chart showing 3 publications over 3 distinct years, with a maximum of 1 publications in 2005 and 2006 and 2016

To see the data from this visualization as text, click here.

Year	Minor Topic	Total
2005	1	1
2006	1	1
2016	1	1

Below are the most recent publications written about "Epistasis, Genetic" by people in Profiles.

Henes M, Kosovrasti K, Lockbaum GJ, Leidner F, Nachum GS, Nalivaika EA, Bolon DNA, Kurt Yilmaz N, Schiffer CA, Whitfield TW. Molecular Determinants of Epistasis in HIV-1 Protease: Elucidating the Interdependence of L89V and L90M Mutations in Resistance. Biochemistry. 2019 09 03; 58(35):3711-3726.

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Lin H, Mueller-Nurasyid M, Smith AV, Arking DE, Barnard J, Bartz TM, Lunetta KL, Lohman K, Kleber ME, Lubitz SA, Geelhoed B, Trompet S, Niemeijer MN, Kacprowski T, Chasman DI, Klarin D, Sinner MF, Waldenberger M, Meitinger T, Harris TB, Launer LJ, Soliman EZ, Chen LY, Smith JD, Van Wagoner DR, Rotter JI, Psaty BM, Xie Z, Hendricks AE, Ding J, Delgado GE, Verweij N, van der Harst P, Macfarlane PW, Ford I, Hofman A, Uitterlinden A, Heeringa J, Franco OH, Kors JA, Weiss S, V?lzke H, Rose LM, Natarajan P, Kathiresan S, K??b S, Gudnason V, Alonso A, Chung MK, Heckbert SR, Benjamin EJ, Liu Y, M?rz W, Rienstra M, Jukema JW, Stricker BH, D?rr M, Albert CM, Ellinor PT. Gene-gene Interaction Analyses for Atrial Fibrillation. Sci Rep. 2016 11 08; 6:35371.

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Jones TI, Parilla M, Jones PL. Transgenic Drosophila for Investigating DUX4 and FRG1, Two Genes Associated with Facioscapulohumeral Muscular Dystrophy (FSHD). PLoS One. 2016; 11(3):e0150938.

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Hines IN, Hartwell HJ, Feng Y, Theve EJ, Hall GA, Hashway S, Connolly J, Fecteau M, Fox JG, Rogers AB. Insulin resistance and metabolic hepatocarcinogenesis with parent-of-origin effects in A?B mice. Am J Pathol. 2011 Dec; 179(6):2855-65.

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Clark IE, Dodson MW, Jiang C, Cao JH, Huh JR, Seol JH, Yoo SJ, Hay BA, Guo M. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin. Nature. 2006 Jun 29; 441(7097):1162-6.

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Xu D, Wang Y, Willecke R, Chen Z, Ding T, Bergmann A. The effector caspases drICE and dcp-1 have partially overlapping functions in the apoptotic pathway in Drosophila. Cell Death Differ. 2006 Oct; 13(10):1697-706.

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Xu D, Li Y, Arcaro M, Lackey M, Bergmann A. The CARD-carrying caspase Dronc is essential for most, but not all, developmental cell death in Drosophila. Development. 2005 May; 132(9):2125-34.

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Choe SK, Sagerstr?m CG. Paralog group 1 hox genes regulate rhombomere 5/6 expression of vhnf1, a repressor of rostral hindbrain fates, in a meis-dependent manner. Dev Biol. 2004 Jul 15; 271(2):350-61.

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