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ArcA-regulated glycosyltransferase lic2B promotes complement evasion and pathogenesis of nontypeable Haemophilus influenzae.
The meningococcal vaccine candidate neisserial surface protein A (NspA) binds to factor H and enhances meningococcal resistance to complement.
The meningococcal vaccine candidate GNA1870 binds the complement regulatory protein factor H and enhances serum resistance.
Blood treatment of Lyme borreliae demonstrates the mechanism of CspZ-mediated complement evasion to promote systemic infection in vertebrate hosts.
Factor H and neisserial pathogenesis.
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Factor H and neisserial pathogenesis.
Welsch JA, Ram S. Factor H and neisserial pathogenesis. Vaccine. 2008 Dec 30; 26 Suppl 8:I40-5.
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PubMed
subject areas
Animals
Antigens, Bacterial
Bacterial Proteins
Blood Bactericidal Activity
Complement Factor H
Complement System Proteins
Gonorrhea
Humans
Ligands
Lipopolysaccharides
Meningococcal Infections
Meningococcal Vaccines
N-Acetylneuraminic Acid
Neisseria gonorrhoeae
Neisseria meningitidis
Species Specificity
authors with profiles
Sanjay Ram MD