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p16Ink4a or p19Arf loss contributes to Tal1-induced leukemogenesis in mice.
Shank-Calvo JA, Draheim K, Bhasin M, Kelliher MA. p16Ink4a or p19Arf loss contributes to Tal1-induced leukemogenesis in mice. Oncogene. 2006 May 18; 25(21):3023-31.
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PubMed
subject areas
Animals
Antigens, CD
Antigens, Neoplasm
Apoptosis
Basic Helix-Loop-Helix Transcription Factors
Cell Cycle
Cell Transformation, Neoplastic
Cyclin-Dependent Kinase Inhibitor p16
DNA
Exons
Gene Deletion
Gene Expression Regulation
Gene Expression Regulation, Leukemic
Genes, p16
Glycoproteins
Immunophenotyping
Leukemia-Lymphoma, Adult T-Cell
Mice
Mice, Transgenic
Neoplasm Proteins
Preleukemia
Protein Binding
Protein Structure, Tertiary
Proto-Oncogene Proteins
Receptor, Notch1
S Phase
T-Lymphocyte Subsets
Transcription Factors
Tumor Suppressor Protein p14ARF
Wnt Proteins
authors with profiles
Michelle A Kelliher PhD
p16Ink4a or p19Arf loss contributes to Tal1-induced leukemogenesis in mice.