"Sodium Channels" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Ion channels that specifically allow the passage of SODIUM ions. A variety of specific sodium channel subtypes are involved in serving specialized functions such as neuronal signaling, CARDIAC MUSCLE contraction, and KIDNEY function.
Descriptor ID |
D015222
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MeSH Number(s) |
D12.776.157.530.400.875 D12.776.543.550.450.875 D12.776.543.585.400.875
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Concept/Terms |
Sodium Channels- Sodium Channels
- Channels, Sodium
- Sodium Ion Channels
- Channels, Sodium Ion
- Sodium Channel
- Channel, Sodium
- Ion Channel, Sodium
- Channel, Sodium Ion
- Sodium Ion Channel
- Ion Channels, Sodium
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Below are MeSH descriptors whose meaning is more general than "Sodium Channels".
Below are MeSH descriptors whose meaning is more specific than "Sodium Channels".
This graph shows the total number of publications written about "Sodium Channels" by people in this website by year, and whether "Sodium Channels" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1995 | 1 | 0 | 1 |
1996 | 1 | 0 | 1 |
1997 | 2 | 0 | 2 |
1999 | 1 | 0 | 1 |
2002 | 0 | 1 | 1 |
2004 | 1 | 0 | 1 |
2005 | 1 | 1 | 2 |
2007 | 0 | 1 | 1 |
2008 | 2 | 0 | 2 |
2009 | 2 | 0 | 2 |
2010 | 2 | 0 | 2 |
2012 | 1 | 2 | 3 |
2015 | 1 | 0 | 1 |
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click here.
Below are the most recent publications written about "Sodium Channels" by people in Profiles.
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Gao S, Xie L, Kawano T, Po MD, Guan S, Zhen M, Pirri JK, Alkema MJ. The NCA sodium leak channel is required for persistent motor circuit activity that sustains locomotion. Nat Commun. 2015 Feb 26; 6:6323.
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Sim S, Antolin S, Lin CW, Lin Y, Lin YX, Lois C. Increased cell-intrinsic excitability induces synaptic changes in new neurons in the adult dentate gyrus that require Npas4. J Neurosci. 2013 May 01; 33(18):7928-40.
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Renaud JM, Hayward LJ. Lessons learned from muscle fatigue: implications for treatment of patients with hyperkalemic periodic paralysis. Recent Pat Biotechnol. 2012 Dec; 6(3):184-91.
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van Zundert B, Izaurieta P, Fritz E, Alvarez FJ. Early pathogenesis in the adult-onset neurodegenerative disease amyotrophic lateral sclerosis. J Cell Biochem. 2012 Nov; 113(11):3301-12.
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Clay JR, Forger DB, Paydarfar D. Ionic mechanism underlying optimal stimuli for neuronal excitation: role of Na+ channel inactivation. PLoS One. 2012; 7(9):e45983.
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Jonsson NN, Cutull? C, Corley SW, Seddon JM. Identification of a mutation in the para-sodium channel gene of the cattle tick Rhipicephalus microplus associated with resistance to flumethrin but not to cypermethrin. Int J Parasitol. 2010 Dec; 40(14):1659-64.
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Tan BH, Pundi KN, Van Norstrand DW, Valdivia CR, Tester DJ, Medeiros-Domingo A, Makielski JC, Ackerman MJ. Sudden infant death syndrome-associated mutations in the sodium channel beta subunits. Heart Rhythm. 2010 Jun; 7(6):771-8.
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Lin CW, Sim S, Ainsworth A, Okada M, Kelsch W, Lois C. Genetically increased cell-intrinsic excitability enhances neuronal integration into adult brain circuits. Neuron. 2010 Jan 14; 65(1):32-9.
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Cheng J, Van Norstrand DW, Medeiros-Domingo A, Valdivia C, Tan BH, Ye B, Kroboth S, Vatta M, Tester DJ, January CT, Makielski JC, Ackerman MJ. Alpha1-syntrophin mutations identified in sudden infant death syndrome cause an increase in late cardiac sodium current. Circ Arrhythm Electrophysiol. 2009 Dec; 2(6):667-76.
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Kelsch W, Lin CW, Mosley CP, Lois C. A critical period for activity-dependent synaptic development during olfactory bulb adult neurogenesis. J Neurosci. 2009 Sep 23; 29(38):11852-8.