 Proto-Oncogene Proteins c-rel
"Proto-Oncogene Proteins c-rel" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cellular DNA-binding proteins encoded by the rel gene (GENES, REL). They are expressed predominately in hematopoietic cells and may play a role in lymphocyte differentiation. Rel frequently combines with other related proteins (NF-KAPPA B, I-kappa B, relA) to form heterodimers that regulate transcription. Rearrangement or overexpression of c-rel can cause tumorigenesis.
| Descriptor ID |
D020569
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| MeSH Number(s) |
D12.776.260.682 D12.776.624.664.700.192 D12.776.660.767 D12.776.930.656
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| Concept/Terms |
Proto-Oncogene Proteins c-rel- Proto-Oncogene Proteins c-rel
- Proto Oncogene Proteins c rel
- c-rel, Proto-Oncogene Proteins
- p68 c-rel
- c-rel, p68
- p68 c rel
- Proto-Oncogene Protein c-rel
- Proto Oncogene Protein c rel
- c-rel, Proto-Oncogene Protein
- Proto-Oncogene Proteins rel
- Proto Oncogene Proteins rel
- c-rel Protein
- Protein, c-rel
- c rel Protein
- p68(c-rel)
- rel Proto-Oncogene Product p68
- rel Proto Oncogene Product p68
- Proto-Oncogene Products c-rel
- Proto Oncogene Products c rel
- c-rel, Proto-Oncogene Products
- c-rel Proteins
- Proteins, c-rel
- c rel Proteins
- rel Proto-Oncogene Proteins
- Proto-Oncogene Proteins, rel
- rel Proto Oncogene Proteins
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Below are MeSH descriptors whose meaning is more general than "Proto-Oncogene Proteins c-rel".
Below are MeSH descriptors whose meaning is more specific than "Proto-Oncogene Proteins c-rel".
This graph shows the total number of publications written about "Proto-Oncogene Proteins c-rel" by people in this website by year, and whether "Proto-Oncogene Proteins c-rel" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 0 | 1 | 1 | | 1998 | 0 | 2 | 2 | | 2002 | 0 | 1 | 1 | | 2005 | 0 | 1 | 1 | | 2009 | 1 | 0 | 1 | | 2015 | 0 | 1 | 1 | | 2016 | 0 | 1 | 1 |
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Below are the most recent publications written about "Proto-Oncogene Proteins c-rel" by people in Profiles.
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Vlantis K, Wullaert A, Polykratis A, Kondylis V, Dannappel M, Schwarzer R, Welz P, Corona T, Walczak H, Weih F, Klein U, Kelliher M, Pasparakis M. NEMO Prevents RIP Kinase 1-Mediated Epithelial Cell Death and Chronic Intestinal Inflammation by NF-?B-Dependent and -Independent Functions. Immunity. 2016 Mar 15; 44(3):553-67.
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Wongchana W, Lawlor RG, Osborne BA, Palaga T. Impact of Notch1 Deletion in Macrophages on Proinflammatory Cytokine Production and the Outcome of Experimental Autoimmune Encephalomyelitis. J Immunol. 2015 Dec 1; 195(11):5337-46.
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Castro I, Wright JA, Damdinsuren B, Hoek KL, Carlesso G, Shinners NP, Gerstein RM, Woodland RT, Sen R, Khan WN. B cell receptor-mediated sustained c-Rel activation facilitates late transitional B cell survival through control of B cell activating factor receptor and NF-kappaB2. J Immunol. 2009 Jun 15; 182(12):7729-37.
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Shin HM, Minter LM, Cho OH, Gottipati S, Fauq AH, Golde TE, Sonenshein GE, Osborne BA. Notch1 augments NF-kappaB activity by facilitating its nuclear retention. EMBO J. 2006 Jan 11; 25(1):129-38.
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Cabatingan MS, Schmidt MR, Sen R, Woodland RT. Naive B lymphocytes undergo homeostatic proliferation in response to B cell deficit. J Immunol. 2002 Dec 15; 169(12):6795-805.
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Lin SC, Wortis HH, Stavnezer J. The ability of CD40L, but not lipopolysaccharide, to initiate immunoglobulin switching to immunoglobulin G1 is explained by differential induction of NF-kappaB/Rel proteins. Mol Cell Biol. 1998 Sep; 18(9):5523-32.
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Yi AK, Krieg AM. CpG DNA rescue from anti-IgM-induced WEHI-231 B lymphoma apoptosis via modulation of I kappa B alpha and I kappa B beta and sustained activation of nuclear factor-kappa B/c-Rel. J Immunol. 1998 Feb 01; 160(3):1240-5.
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Lin SC, Stavnezer J. Activation of NF-kappaB/Rel by CD40 engagement induces the mouse germ line immunoglobulin Cgamma1 promoter. Mol Cell Biol. 1996 Sep; 16(9):4591-603.
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